Search results for " thalassemia"

showing 10 items of 34 documents

Hematopoietic peripheral circulating blood stem cells as an independent marker of good transfusion management in patients with β-thalassemia: results…

2016

Background Beyond hemoglobin (Hb) levels and performance status, further surrogate markers of appropriate transfusion management should improve the quality of thalassemia care. We investigated the levels of peripheral circulating CD34+ stem cells as an independent marker of appropriate hematopoietic balance in patients with thalassemia. Study design and methods Peripheral circulating CD34+ stem cells, colony-forming unitgranulocyte, erythrocyte, macrophage, magakaryocyte (CF-GEMM), colony-forming unitgranulocyte/macrophage (CFU-GM), and erythroidburst-forming units (BFU-E) were assayed, according to standard procedures. Patients with thalassemia major (TM) and thalassemia intermedia (TI) we…

0301 basic medicinemedicine.medical_specialtyThalassemiamedicine.medical_treatmentImmunologyCD34Hematopoietic stem cell transplantationGastroenterology03 medical and health sciences0302 clinical medicineInternal medicinemedicineImmunology and Allergybusiness.industryBeta thalassemiaHematologymedicine.diseaseRed blood cellHaematopoiesis030104 developmental biologymedicine.anatomical_structureImmunologyHemoglobinStem cellbusiness030215 immunologyTransfusion
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Fever of Unclear Origin and Cytopenia Because of Acute Splenic Sequestration in a Young Immunocompetent Carrier of Beta-Globin Mutation for Hb Vallet…

2008

Fever of unclear origin is a clinical challenge in medical practice. Infectious diseases, neoplasms, and collagen vascular illnesses are its main causes in adults and children. Acute splenic sequestration crises, a known potentially fatal complication of sickle cell disease and sickle beta-thalassemia, are uncommon in beta-heterozygosis. We describe a case of prolonged recurrent episodes of fever with spontaneous resolution, commencing at age 10 in a 15-year-old boy with a history of hypochromic microcytic anemia attributed to a thalassemic trait. He was admitted twice to our university hospital for continuous-remittent fever with a pruritic, macular evanescent Still's skin rash, severe spl…

AdultMaleAdolescentFeverAnemiaHemoglobins AbnormalThalassemiaHepatosplenomegalybeta-Globinshemic and lymphatic diseasesmedicineHumansChildCytopeniabusiness.industryBeta thalassemiaAnemiaGeneral Medicinemedicine.diseaseHematologic DiseasesHypochromic microcytic anemiaHemoglobinopathyMutationImmunologymedicine.symptomSplenic diseasebusinessImmunocompetenceSpleenThe American Journal of the Medical Sciences
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Serum and fecal pancreatic enzymes in beta-thalassemia major

1997

This study, using indirect tests, demonstrated that exocrine pancreatic function is impaired in a proportion of patients with beta-thalassemia major (TM), though this impairment is generally mild or moderate.Impaired structure and function of the exocrine pancreas has been reported in patients with Beta-thalassemia major.In this study we measured fecal fats and serum and fecal pancreatic enzymes in 30 patients (13 M, 17 F) with TM, mean age 22.1 yr (range 14-39) and compared them with those of a matched group of healthy controls. Results were correlated with age, serum ferritin, blood transfusion, and various nutritional parameters. Enzymes assays included: serum pancreatic amylase (PA), li…

AdultMaleHemolytic anemiamedicine.medical_specialtyPancreatic diseaseAdolescentHydrolasesGastroenterologyFatsPathogenesisFecesEndocrinologyInternal medicinemedicineChymotrypsinHumansTrypsinFeceschemistry.chemical_classificationPancreatic Elastasebusiness.industrybeta-ThalassemiaGastroenterologyBeta thalassemiaLipasemedicine.diseaseHemoglobinopathyEnzymeEndocrinologyOncologychemistryAmylasesExocrine Pancreatic InsufficiencyFemalebusinessPancreatic enzymesInternational Journal of Pancreatology
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Evaluation of the efficacy of oral deferiprone in beta-thalassemia major by multislice multiecho T2*.

2006

Objectives: Oral deferiprone (L1) appears to be promising in the treatment of beta-thalassemia major (TM) patients. T2* magnetic resonance imaging (MRI) with a single measurement in the mid-ventricular septum was validated as a quantitative evaluation of myocardial iron overload. Previous studies suggested a marked heterogeneity of iron distribution in the myocardium. We set up a multislice multiecho T2* MRI for the detection of this heterogeneity. The aim of our study was to investigate differences between the L1 vs. the subcutaneous desferrioxamine (DF)-treated patients using this new approach.Methods: Thirty-six beta-TM patients (age 29 +/- 8 yr) underwent MRI. Eighteen patients received…

AdultMaleIron OverloadPyridonesCoefficient of variationDeferoxamineBETA THALASSEMIA MAJORchemistry.chemical_compoundMagneticsmultislice multiecho T2*MedicineHumansMultisliceDeferiproneReproducibilitymedicine.diagnostic_testbusiness.industryMyocardiumbeta-ThalassemiaBeta thalassemiaMagnetic resonance imagingHematologyGeneral MedicineMiddle Agedmedicine.diseaseMagnetic Resonance Imagingchelation treatmentmedicine.anatomical_structurechemistryVentricleFemalebusinessNuclear medicineDeferiproneEuropean journal of haematology
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Transfusional Hemochromatosis: Quantitative Relation of MR Imaging Pituitary Signal Intensity Reduction to Hypogonadotropic Hypogonadism

2000

To assess the relationship between magnetic resonance (MR) imaging pituitary signal intensity reduction in patients with transfusional hemochromatosis and the clinical manifestation of hypogonadotropic hypogonadism.Pituitary MR imaging at 0.5 T was performed in 38 consecutive patients affected by secondary hemochromatosis and in 20 healthy volunteers. Serum ferritin levels were estimated in the affected population. Twenty (53%) of the 38 patients had hypogonadotropic hypogonadism diagnosed. Pituitary-to-fat signal intensity ratios were calculated from coronal gradient-echo (GRE) T2*-weighted MR images. The relationship between the quantitative reduction of the pituitary-to-fat signal intens…

AdultMalePathologymedicine.medical_specialtyPituitary glandAdolescentHemochromatosiPopulationSensitivity and SpecificityPituitary Gland AnteriorHypogonadotropic hypogonadismmedicineHumansBlood TransfusionRadiology Nuclear Medicine and imagingChildeducationHemochromatosisFerritineducation.field_of_studymedicine.diagnostic_testbusiness.industryHypogonadismbeta-ThalassemiaTransfusion ReactionBeta thalassemiaMagnetic resonance imagingmedicine.diseaseMagnetic Resonance Imagingmedicine.anatomical_structureROC CurveCoronal planeFerritinsFemaleHemochromatosisbusinessNuclear medicineHumanHormoneRadiology
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Quantification of HBG mRNA in primary erythroid cultures: prediction of the response to hydroxyurea in sickle cell and beta-thalassemia

2013

Background and Objective Increased expression of fetal hemoglobin (HbF) may ameliorate the clinical course of hemoglobinopathies like sickle cell disease (SCD) and β-thalassemia. Hydroxyurea (HU) can stimulate HbF production in these diseases but the response is highly variable indicating the utility of developing an in vitro test to predict the patient's response to HU. We assessed whether the HbF response of patients with SCD and thalassemia intermedia (TI) to HU correlates with HBG (both γ-globin genes) expression in their cultured erythroid progenitors following exposure to HU. Patients and Methods We exposed primary erythroid cultures from peripheral blood mononuclear cells from 30 pat…

AdultMalecongenital hereditary and neonatal diseases and abnormalitiesCellPrimary Cell CultureGene ExpressionAnemia Sickle CellBiologyPeripheral blood mononuclear cellhydroxyurealiquid erythroid cultureYoung AdultIn vivohemic and lymphatic diseasesFetal hemoglobinmedicineHumansgamma-GlobinsRNA MessengerFetal HemoglobinAgedErythroid Precursor CellsMessenger RNAbeta-ThalassemiaBeta thalassemiaHematologyGeneral MedicineMiddle Agedmedicine.diseaseb-thalassemiaMolecular biologyReal-time polymerase chain reactionmedicine.anatomical_structureTreatment OutcomeCell cultureFemalesickle cell disease
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Standardized T2* map of normal human heart in vivo to correct T2* segmental artefacts.

2007

A segmental, multislice, multi-echo T2* MRI approach could be useful in heart iron-overloaded patients to account for heterogeneous iron distribution, demonstrated by histological studies. However, segmental T2* assessment in heart can be affected by the presence of geometrical and susceptibility artefacts, which can act on different segments in different ways. The aim of this study was to assess T2* value distribution in the left ventricle and to develop a correction procedure to compensate for artefactual variations in segmental analysis. MRI was performed in four groups of 22 subjects each: healthy subjects (I), controls (II) (thalassemia intermedia patients without iron overload), thala…

AdultMalemedicine.medical_specialtyheart; thalassemia; MRI; heart iron overload; multislice multi-echo T2*Segmental analysisT2 mappingGroup iiheartSensitivity and SpecificityT2* mapVentricular Dysfunction LeftReference ValuesIn vivoImage Interpretation Computer-AssistedmedicineHumansRadiology Nuclear Medicine and imagingMultisliceSpectroscopyheart iron overloadbusiness.industryMyocardiumHealthy subjectsReproducibility of ResultsHuman heartImage Enhancementmultislice multi-echo T2*Magnetic Resonance ImagingSurgerymedicine.anatomical_structuresegmental artefactVentricleThalassemiaMolecular MedicineFemaleArtifactsNuclear medicinebusinessMRI
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Inhibition of Fibroblast Growth Factor-23 (FGF-23) Rescues Bone and Hematopoietic Stem Cell Niche Defects in Beta-Thalassemia, Uncovering the Missing…

2021

Abstract The bone marrow (BM) niche regulation and interactions with hematopoietic stem cells (HSC) have been extensively studied in steady state conditions and malignancies, but are still underexplored in hematological inherited disorders. We provided the first demonstration of impaired HSC function caused by an altered BM niche in a non-malignant disease, beta-thalassemia (BT) (Aprile et al., Blood 2020). BT is a globally widespread congenital hemoglobin disorder, resulting in severe anemia, ineffective erythropoiesis and multi-organ secondary complications, including bone alterations. Correction of the genetic defect is achieved by transplantation of HSC from healthy donors or autologous…

Fibroblast growth factor 23HaematopoiesisHematopoietic stem cell nicheImmunologymedicineBeta thalassemiaCell BiologyHematologyBiologymedicine.diseaseBiochemistryCell biologyBlood
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Other Genetic Chaperonopathies

2013

In this chapter are presented chaperonopathies in which a genetic mechanism is involved but are different from those discussed in chapter 4. Thus, chaperonopathies due to gene dysregulation such as those observed in aged individuals and in some cases with neurodegenerative diseases (e.g., Alzheimer’s, Huntington’s, Parkinson’s, and other conditions), are presented. Likewise, examples of the impact of chaperone-gene polymorphisms on health and disease are given. The quantitative chaperonopathies attributable to gene dysregulation are discussed.

GeneticsMechanism (biology)medicineBeta thalassemiaDiseaseBiologymedicine.diseaseGene
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Three Distinct Groups of Phenotype Severity in Beta-Thalassemia

2020

Background Thalassemia Syndromes (TS) are commonly classified as transfusion-dependent-thalassemia (TDT) or non-transfusion-dependent thalassemia (NTDT) at diagnosis on the basis of requirement for lifelong regular transfusion therapy for survival. However, data from observational studies and expert opinion suggest that these categories may reflect a wide spectrum rather than a dichotomy, and may actually be interchangeable at many parts of the disease journey. Thus, an evaluation of alternate clusters to classify TS patients remains of merit. Aims The aim of this study was to cluster TS patients on the basis of possible clinical indicators of phenotype severity (IPhS) using suitable algori…

ImmunologyImmunologymedicineBeta thalassemiaCell BiologyHematologyBiologymedicine.diseaseBiochemistryPhenotypeBlood
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